345 Ground Zero workers have died of cancer and other CANCER-STRICKEN Ground Zero workers have finally received a compensation checks – for zero dollars.

The city recently settled lawsuits by 10,000 WTC workers, more than 600 of whom have developed cancer. But officials have so far insisted there is no scientific proof that Ground Zero smoke and dust caused cancer.

An FDNY spokesman gave a statement for Dr. Prezant, saying: ‘The study is ongoing, and no conclusions have been reached on whether cancer rates have increased for firefighters.’ But fire union bosses in New York have expressed their concern about the findings.

Al Hagan, head of the fire-officers union, told the New York Post: ‘I’m led to believe that the numbers for those cancers across all ranks in the Fire Department of people who worked at Ground Zero is up significantly, and we’re all very concerned about it, as are our families.’ Steve Cassidy, president of the firefighters union, said Ground Zero’s ‘toxic stew’ has proven lethal. He said: ‘It’s a fact that New York City firefighters are dying of cancer in record numbers. ‘We have buried 10 firefighters in just the last 15 weeks, seven with cancer. On Sept. 10, 2001, they were young, healthy firefighters.’

In 2007, doctors at Mt. Sinai Medical Center, which monitors World Trade Center rescue workers, noted blood cancers like multiple myeloma, which normally strikes in the 60s or 70s, were being found in relatively young officers.

The New York state Health Department has confirmed that 345 Ground Zero workers have died of various cancers as of June 2010.

http://www.dailymail.co.uk/news/article-1373108/9-11-fi…
———

Are these cancers all from toxins in the dust? You know what else causes high rates of cancer?

Every potential client considering a contingent fee agreement with a lawyer should first consider this story about a worker who spent hundreds of hours cleaning up the “ground zero” site in New York City after the 9/11/01 terror attacks.  He was steered to a NY law firm which is handling 10,000 such claims, so he probably assumed they knew what they were doing.  They twisted his arm to get him to accept a settlement for just $10K, all of which went to the firm’s fees, unitemized firm expenses, and paying back a small amount of his workman’s compensation benefits, leaving him, literally, with a check for $0.00, totally broke, and now diagnosed with life-threatening cancer:

CANCER-STRICKEN Ground Zero worker Edgar Galvis has finally received a compensation cheque – for zero dollars.

The man … was relieved to get a cheque in the mail for his court settlement with Merrill Lynch, whose offices he had cleaned.

But he was stunned when he saw the amount: $0.00.

His award had been $10,005, but his lawyers at the firm Worby, Groner, Edelman & Napoli Bern lopped off $2579 for unitemised legal expenses.

Then they took a 33.3 per cent fee of $2124. They also subtracted $352, a fee to the lawyer who referred him.

The remaining $4950 was withheld for unspecified “liens”, the letter says.

Mr Galvis thinks this was repayment of workers’ compensation for aid.

…”I couldn’t believe it. I thought it was a joke.”…In May 2005, a friend gave him a business card passed out by the law firm. A representative came to his home.

“The man told me that more than likely I will get sick and I would get 60 per cent of whatever he won,” Mr Galvis said.

“He even mentioned the words ‘millions of dollars’.”

In April 2010, he got a $10,000 offer. A letter from the law firm said he could expect about $5000 after expenses and fees.

It warned that if his case went to trial and he lost, he could owe the firm up to $100,000 in costs.

He took the settlement.  [Apparently the settlement was based on losing sleep and sinus problems, but then he was diagnosed with cancer, but the firm told him] it was “too late” to adjust his claim.  [Sounds odd to me.]

The total Merrill settlement came to $18 million for about 400 clients, documents show.

Galvis is one of nearly 10,000 Ground Zero workers represented by Napoli Bern, which led talks for a separate settlement with the city for $712 million. A partner in the firm, Paul Napoli, did not respond to a request for comment.

Lawyers working on a contingent fee basis can’t make money spending lots of time on small claims, which is what they thought this was when they settled it.  (There’s even a chance the expenses aren’t real, just another profit center.)  Even though they knew the client might get sicker — they even predicted it — they sold him out, pressured him to settle, and apparently didn’t make any effort to amend or restart the proceedings to protect his interests once he got sicker.  Settling for $10K, given his financial situation, even if he got to keep the whole $10K, wouldn’t solve any of his problems — but for the firm, assuming they spent little or no time on a matter, you can make $50 million if you make roughly $5K each on 10K cases.

A particularly dirty tactic was to threaten the client with an absurdly inflated amount for expenses to go to trial — not something they mentioned up front, apparently.  The primary expense in this sort of case is usually expert medical testimony, but this shouldn’t cost anything close to $100K and it wouldn’t make sense for the firm to recommend settlement without already having an expert opinion.  More reasonable contingent fee lawyers eat the expenses rather than bankrupt the clients they fail to help.  (This is one of the firms that also earned international disdain for the amount of money they demanded for “legal fees” from the government fund created to help the first responders, etc., who took well over half the money — including payments to government lawyers — and didn’t try a single case.  I gather they’ve been paid hundreds of millions of dollars and haven’t even come close to trying a single case.)

Lawyers doing contingent fee work often resort to the same sorts of tactics to woo clients that are used by used car salespeople and con men.  This happens every day with many of the contingent fee mills.  You need to shop around, make a record of what they tell you before and after you sign, and complain to the police and bar if you believe you’ve been taken advantage of.

In this case, the plaintiff’s lawyer rendered a far more valuable service to Merrill Lynch, which dodged a multi-million dollar bullet thanks to their opponent’s professional advisers.  We’ll have to see whether the authorities in New York decide to step in — not likely based on their track record.

Australian Story: Cancer-stricken Ground Zero worker receives compensation cheque for 0$.

American Hero and Army Sergeant Who Gave Life To Save Afghan Child Being Flown Home For Burial

This section Copied from: http://infofeeder.info

This should be getting the attention 
Friday, 30 March 2012 02:39
I’m not really looking for debate on this as much as I want to make an effort to educate people. Sure, the
military has screwed up in Afghanistan. We’ve pissed on bodies, burnt books, and one man has even
committed pre-meditated mass murder. But we’re not always bad. For every one of those stories you
hear, much more like this happen. Its not a matter of foreign relations or “winning hearts and minds”.
Its about caring for your fellow human being, regardless of nationality, race, sex, etc. This man should
be glorified for the hero he is. When I read about fellow brothers doing things like this, I tear up everytime.
I don’t care what anyone says, this generation of service member is the “greatest generation”.
Army Sergeant Who Gave Life To Save Afghan Child Being Flown Home For Burial | Fox News

http://www.debatepolitics.com/breaking-news-mainstream-media/122508-should-getting-attention.html

Army sergeant who gave life to save Afghan child being flown home for burial

Published March 29, 2012

| FoxNews.com


An Army sergeant and father of three from Rhode Island who gave his life to save an Afghan child from being run over by a 16-ton armored fighting vehicle is being flown back to the U.S. and will be buried Monday.

Sgt. Dennis Weichel, 29, died in Afghanistan last week after he dashed into the path of an armored fighting vehicle to scoop up the little girl, who had darted back into the roadway to pick up shell casings, according to the Army. Weichel, a Rhode Island National Guardsman, was riding in the convoy in Laghman Province in eastern Afghanistan when he jumped out to save the girl, who was unhurt.

  • Dennis Weichel 1

    This image, obtained from WPRI.com, shows 29-year-old Sgt. Dennis Weichel.

  • Dennis Weichel 2
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“He would have done it for anybody,” Staff Sgt. Ronald Corbett, who deployed with Weichel to Iraq in 2005, said in a quote posted on the U.S. Army website. “That was the way he was. He would give you the shirt off his back if you needed it. He was that type of guy.”

The child was one of several who were collecting the casings, which can be sold and recycled in Afghanistan. Weichel and other soldiers in the convoy got out of their vehicles to shoo the kids from danger as the heavy trucks bore down. But the girl ran back onto the road as a MRAP, or Mine Resistant Ambush Protected Vehicle, approached. Weichel swung the girl to safety but was run over and later died from his injuries at Jalalabad Medical Treatment Facility, according to a press release from the Rhode Island National Guard.

Weichel, who had been a member of the Rhode Island National Guard since 2001, had arrived in Afghanistan a few weeks ago. He was a member of C Company, 1st Battalion, 143 Infantry. Weichel was previously deployed to Iraq in 2005 as a member of 3/172 Det 2 Mountain Infantry.

Weichel, who lived in Providence and was engaged to be married, leaves his parents, fiancee and three young children. His body is scheduled to be flown to Dover Air Force Base in Delaware on Saturday. Weichel will be buried in Rhode Island Veterans Cemetery in Exeter.

“Tragically, Spc. Weichel has made the supreme sacrifice and at this time, we are mindful of the impact of that sacrifice on his family and friends,” said Maj. Gen. Kevin McBride, adjutant general of the Rhode Island National Guard, in a written statement. “We leave no Soldier behind…. and we will not leave Spc. Weichel’s family behind.”

Read more: http://www.foxnews.com/us/2012/03/29/army-sergeant-who-gave-life-to-save-afghani-child-being-flown-home-for-burial/#ixzz1qZ7ZjwzY

Could the LeRoy 12 Mystery Epidemic Be Sensationalized by the N.Y. Media?

 Post Streptococcal Autoimmune Neurobehavioral Disorder

AKA

(( Neuro ‘Psychiatric’ Disorder))

 conversion/ fake disorder

“This is not a mystery, it’s Post Streptococcal Autoimmune Neurobehavior Disorder. It can occurs -anytime even a year or two later- post having an infection, from the TOXINS of various strains of strep, depending upon an individuals immune system. Symptoms can come and go in the future, depending upon if an person with it becomes run down and their immune system compromised. Furthermore, the medical staff and school staff are well aware of this but have chosen to ignore it, preferring instead to sensationalize it in the media, that’s how I feel, as my son was diagnosed with this over 12 years ago and the NY State was sent his medical records. It’s downright foolish and dangerous to ignore this as health complications from strep can actually be fatal.”Deanna Bruce

Tourette syndrome and other tic disorders. Schemat
Deanna Bruce 

Thanks Daniel & Elbee, One if the biggest problems that I can see- is that what was once called a Neurobehavior Disorder-years earlier has somehow now been coined a neuro ‘psychiatric’ disorder. An example of a reason-why it is so important to know when a person has a Post Strep movement disorder – is that; for example what happened to a boy on my son’s hockey team -if a doc ever wrongly prescribes a stimulant i.e. ritalin for ADHD- that person can have life long devastating effects…seizures, limbs twisted in contortions. Left untreated- Strep can cause more than rheumatic fever. My son a couple of years ago -got a bit run down, and missed sleep playing soccer- yet, with a negative for strep throat swab, he went into kidney failure- later a blood titre 300 times too high -showed it actually was Strep related. Watch someone suffer in horrific pain, on a palliative unit- and the very thought of this being called a conversion/ fake disorder– is disgusting.
Deanna Bruce

As mentioned previously, the New York State Department of Health was given a medical record copy to prove what I am saying of the Strep titre / Neurologist who saw my son re: PANDAS 12 years ago/ and the kidney failure…..one would assumethe ‘right’ people have already seen it. The kids ought to be given an antibiotic at any rate….in case it helps their symptoms subside some. Personally, I’ve about given up trying to make it easy for Specialists there who are supposed to already know all this.

The Pediatric Autoimmune Neuropsychiatric Disorders Associated With Streptococcal Infection (PANDAS) Etiology for Tics and Obsessive-Compulsive Symptoms: Hypothesis or Entity? Practical Considerations for the Clinician

  1. Roger Kurlan, MD*,
  2. Edward L. Kaplan, MD

+Author Affiliations


  1. *Cognitive and Behavioral Neurology Unit, Department of Neurology, University of Rochester School of Medicine, Rochester, New York

  2. Department of Pediatrics, World Health Organization Collaborating Center for Reference and Research on Streptococci, University of Minnesota School of Medicine, Minneapolis, Minnesota

ABSTRACT

Clinicians have been faced with much publicity and contradictory scientific evidence regarding a recently described condition termed pediatric autoimmune neuropsychiatric disorders associated with streptococcal infection (PANDAS). It has been proposed that children with PANDAS experience tics, obsessive-compulsive behavior, and perhaps other neuropsychiatric symptoms as an autoimmune response to streptococcal infection. We review current scientific information and conclude that PANDAS remains a yet-unproven hypothesis. Until more definitive scientific proof is forthcoming, there seems to be insufficient evidence to support 1) routine microbiologic or serologic testing for group A streptococcus in children who present with neuropsychiatric symptoms or 2) the clinical use of antibiotic or immune-modifying therapies in such patients. The optimum diagnostic and therapeutic approach awaits the results of additional research studies.

Key Words:

With a description of 50 patients, Swedo et al1 proposed a hypothesis that childhood obsessive-compulsive disorder (OCD) and/or tics may arise as a result of a poststreptococcal autoimmune process; the authors suggested the acronym PANDAS (pediatric autoimmune neuropsychiatric disorders associated with streptococcal infections). It additionally was suggested that, by a process of molecular mimicry, somatic epitopes of the group A β-hemolytic streptococcus (Streptococcus pyogenes) (GABHS) evoke antibodies that are capable of cross-reacting with specific areas of the human brain (eg, the basal ganglia) to produce neuropsychiatric and behavioral symptoms. The authors hypothesized that this condition might be a forme fruste of the pathogenetic process thought to underlie Sydenham’s chorea and therefore also may be related to rheumatic fever.2,3 The observation that Sydenham’s chorea includes tics4 and sometimes obsessive-compulsive symptoms5contributed to the proposal that a poststreptococcal autoimmune mechanism(s) might lead to a more diverse neurobehavioral symptom spectrum. Subsequently, the clinical spectrum associated with this constellation of neuropsychiatric signs and symptoms (PANDAS) was proposed to include a number of additional manifestations including attention-deficit/hyperactivity disorder,6 myoclonus,7 dystonia,8 paroxysmal dyskinesias,9 acute disseminated encephalomyelitis,10 and anorexia nervosa.11

This PANDAS hypothesis has stimulated clinical and basic research and has resulted in considerable clinical and scientific controversy1214; the controversy has influenced clinical care. Because of the implications for the primary care physician, the neurologist, the psychiatrist, the infectious disease specialist, the cardiologist (if it is to be considered part of the rheumatic fever complex, as is Sydenham’s chorea), and even the basic scientist, it is appropriate to objectively examine available information regarding the hypothesis and to provide recommendations for a current, optimal clinical approach to patients suspected of having PANDAS.

THE DIAGNOSTIC CRITERIA: IS PANDAS AN ADEQUATELY DEFINED CLINICAL DIAGNOSIS?

Five criteria were proposed to define the PANDAS hypothesis1 (see Table 1).

TABLE 1.

National Institute of Mental Health Clinical Diagnostic Criteria for PANDAS

Criterion 1

As mentioned, several authors have suggested that tics and OCD may not be the only clinical features of PANDAS, and thus the true boundaries of the PANDAS clinical spectrum remain to be scientifically validated or standardized. Even considering only tics and OCD, the level and characteristics of symptom severity required for diagnosis have not been defined yet by controlled studies.

Criterion 2

The proposed age at onset, ranging between 3 years and the beginning of puberty, may be too arbitrary and may simply be the result of referral patterns for the 50 initially described patients. For example, the validity of the proposed age-at-onset criterion is raised by the report of a 25-year-old with sudden-onset OCD after pharyngitis.15 Furthermore, because the vast majority of typical Tourette’s syndrome (TS) manifestations (ie, tics) begin during the prepubertal period (75% having onset before 11 years old16), the age-at-onset criterion is not sufficiently specific in distinguishing PANDAS from the more established diagnostic criteria for TS.17 Recognizing a documented higher likelihood of exposure of schoolchildren to GABHS, no pathogenetic or immunologic basis has been confirmed to explain why the onset might be expected to include only young children.

Criterion 3

A unique specificity of a clinical course consisting of abrupt onset or dramatic exacerbations has not been documented adequately. With careful observation, tics are either present or not; thus, the onset (or parental awareness) of tic disorders is not gradual. Often with a sudden recognition of tics by parents, in retrospect a prior history of milder tics can be elicited, making the precise timing of clinical onset confusing. Experienced neurologists and psychiatrists are aware that tic disorders and OCD characteristically wax and wane spontaneously, with exacerbations followed by remissions. The degree of clinical change required for recognizing exacerbations to distinguish a “PANDAS-like process” from the typical course of more established tic and OCDs has not been differentiated or quantitated adequately. For example, 1 recent study of 80 consecutively examined (unselected) patients in a tic-disorders clinic found that 53% reported sudden, explosive onset or worsening of their tics.18 These patients were not considered to have met criteria for a diagnosis of PANDAS. Preliminary evaluation of our own ongoing, case-control epidemiologic study of PANDAS indicated that 36% of the carefully selected control subjects (who had no recognized link between symptoms and GABHS infection) also reported an abrupt onset or dramatic exacerbations (R. Kurlan, MD, unpublished data, 2003). Thus, clinical course does not seem particularly useful in distinguishing patients suspected of PANDAS from children with more typical cases of TS or OCD.

Criterion 4

Onset or exacerbations of neuropsychiatric symptoms temporally related to GABHS infection has been proposed as the sine qua non of the diagnosis of PANDAS. Yet, despite limited studies to establish this, it has not been documented adequately in prospective studies and remains 1 of the most controversial aspects of the hypothesis. GABHS infections are very common, and the prevalence of this organism in the upper respiratory tract of asymptomatic school children (“carriers”) often is relatively high.19 This epidemiologic phenomenon of high prevalence of GABHS has resulted in a number of other disease entities (among the best examples are Henoch-Schönlein purpura and Kawasaki disease), which also have been proposed to be associated with group A streptococcal infections. However, when adequately controlled studies were conducted, streptococci were eliminated as the causal factor.20 The confusion about the role of GABHS may be promoted further because, clinically, it is appreciated that tic disorders and OCD worsen during times of stress or illness of any kind. The worsening of symptoms might be expected with streptococcal upper respiratory tract infection and could even occur in the relatively large percentage of children who are asymptomatic streptococcal carriers if simultaneous stressful situations occur (from any other cause).

Additionally, review of published studies indicates that the temporal boundaries of a proposed “temporal” relationship with streptococcal infections have never been established. In Sydenham’s chorea, often cited as the model for PANDAS, the choreoathetoid movements typically appear 3 to 5 months after GABHS infection. Swedo et al1 implied that an infection up to 9 months before symptom onset may be acceptable for the diagnosis of PANDAS but also have indicated that the presence of GABHS in the upper respiratory tract (not differentiating infection from the carrier state) may not be detectable until weeks after onset or exacerbation. Such a lack of precision does not assist in establishing a distinct syndrome.

Thus, the absence of regular, continuous, prospective throat cultures along with streptococcal antibody determinations has made it impossible to establish conclusively a temporal relationship between streptococcal infection and onset or exacerbation of neuropsychiatric symptoms. Sufficiently long, longitudinal, prospective studies conclusively demonstrating streptococcal infection and correlating this with clinical findings are required.

Criterion 5

The final diagnostic criterion, that there must be abnormalities on neurologic examination, presents a difficult and practical clinical problem. For example, the 1998 report that “choreiform” movements are often present in patients in whom a diagnosis of PANDAS is considered indicates that the cohort on which the proposed diagnostic criteria were based may well have included subjects with mild chorea that might also be compatible with more conventional Sydenham’s chorea.1

CLINICAL MANAGEMENT OF PATIENTS HAVING SOME OR ALL OF THESE NEUROPSYCHIATRIC MANIFESTATIONS

Laboratory Testing

An initial concern of pediatricians and primary care physicians about clinical management of patients with this symptom complex is which, if any, diagnostic laboratory studies should be done. Given the current status of as-yet incompletely defined support for the existence of a specific entity called PANDAS, the physician may be cautious in deciding whether to obtain throat cultures or determine antistreptococcal antibodies in all children presenting with neurobehavioral symptoms such as tics, obsessions, or compulsions. Because of potential difficulties in interpretation of single tests, even with “positive” culture results, the high prevalence of so many GABHS carriers in normal populations potentially confuses the issue.

Patients with signs and symptoms compatible with streptococcal upper respiratory tract infections should be studied for the presence of GABHS just as with any other patient. In the absence of symptoms of streptococcal infection, the relevance of a positive culture is difficult to interpret. The many uncertainties encountered in interpreting streptococcal antibody titers in otherwise normal children are recognized.21 Therefore, to use streptococcal antibody tests or throat cultures in an asymptomatic child either as a screening test or to confirm a diagnosis of PANDAS is problematic.

Two research laboratory tests, measurement of circulating antineuronal antibodies and an assay for the presence of an alleged rheumatic fever-associated T lymphocyte alloantigen called D8/17,22 have not been able to correlate reliably and consistently with suspected PANDAS cases.14

Although radiologically suspected enlargement of caudate nuclei has been described in some suspected patients, routine magnetic resonance imaging cannot be recommended currently, because the validity has not been substantiated.23

Therapy

What treatment should be considered for patients who present with the manifestations associated with the PANDAS constellation of symptoms?

Neuropsychiatric Drugs

Children with disabling tics or OCD should receive appropriate treatment for their symptoms, including medications (eg, tic suppressants such as α-agonists, classical or atypical antipsychotics, or antiobsessional drugs such as selective serotonin-reuptake inhibitors) or nonpharmacologic approaches (eg, cognitive behavioral therapy for OCD).24

Antibiotics to Prevent Strep Infections

The only published clinical trial of prophylactic penicillin in children with this syndrome revealed no conclusive evidence that the antibiotic reduced clinical exacerbations.25 Admittedly, the duration of that study was too short to allow definite conclusions. A later report suggesting improvement of new-onset or acute exacerbations of symptoms in such children with antibiotics provides inadequate support for such an approach, because treatment was not placebo-controlled and was unblinded.26 It is well known in treatment studies of TS that there is a substantial placebo effect; the natural course of TS and OCD is such that exacerbations are followed by remissions. This latter phenomenon of “reversion to the mean” implies that virtually any intervention at the time of peak symptoms may seem successful. Only a double-blind, placebo-controlled study can identify a true therapeutic effect.

Another reason to feel comfortable with avoiding antibiotic treatment for these patients is that, to date, no cases have been reported to develop any rheumatic carditis as occurs in patients with Sydenham’s chorea. This is a very important point to remember, because some have attempted to relate this syndrome to rheumatic fever. It is recognized that as many as one third of patients with Sydenham’s chorea ultimately will have evidence of rheumatic valvular heart disease.27 This issue requires additional investigation, because the lack of heart disease strongly argues against a relationship between PANDAS and Sydenham’s chorea or other forms of rheumatic fever.

IS THERE A CASE FOR IMMUNOMODULATORS?

Because the postulated mechanism for a PANDAS syndrome implies an immunologic dysfunction, therapeutic trials have attempted to modify the effects of an immune response. A published study involving 29 patients fulfilling suggested criteria for PANDAS reported sustained benefits both after plasma exchange and in patients after intravenous immunoglobulin.28However, Singer,29 in an accompanying commentary, pointed out numerous methodologic concerns about the trial including highly selective recruitment, small sample size, lack of severity matching within treatment groups, limited comparisons with controls, absence of sham apheresis, concomitant use of psychotropic medications, possible treatment-order effects, and adverse effects that occurred in approximately two thirds of subjects receiving active treatment. A subsequent note of caution was issued in 2000 by the National Institute of Mental Health recommending that, at this time, plasma exchange and intravenous immunoglobulin be reserved only for subjects participating in approved research protocols because there are distinct risks involved.30 To date, there are no additional published data to encourage use of these immune-modifying techniques.

CONCLUSIONS

The current state of knowledge dictates that the concept of a constellation of neuropsychiatric signs and symptoms that are associated with GABHS infections (the PANDAS hypothesis) should be considered only as a yet-unproved hypothesis. History reminds us that the same autoimmune hypothesis for childhood autism, including poststreptococcal elements,31 has been pursued for decades32 without conclusive proof. The PANDAS hypothesis is the focus of clinical and laboratory research. There currently is insufficient evidence to support routine laboratory testing or antibiotic/immune-modifying treatment for suspected cases. Although there is great need for a more effective diagnostic and therapeutic approach for these children, the clinician is obliged to await additional data from well-controlled prospective studies to determine whether PANDAS is a documentable entity or will remain an unproven hypothesis.

ACKNOWLEDGMENTS

This work was supported by National Institutes of Health research grant NS42240 from the National Institute of Neurological Disorders and Stroke.

OCD, obsessive-compulsive disorder • PANDAS, pediatric autoimmune neuropsychiatric disorders associated with streptococcal infection • GABHS, group A β-hemolytic streptococcus • TS, Tourette’s syndrome

REFERENCES

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